Contact Information

Department Chair
Daniel Shain

Department Secretary
Janet Caruso
(856) 225-6497

Undergraduate Biology
Kwangwon Lee

Graduate Biology
William M. Saidel

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Student Spotlight

We R Rutgers-Camden: David Luor

David Luor"I am becoming someone who likewise engages meaningfully with the world. Thank you, Rutgers-Camden." Read more ...

Biology Seminar Series

Upcoming Seminars

“Extracellular and intracellular ATP concentration in Neurospora, mouse, and rat: metabolic circadian/ultradian rhythm markers?”


Steven Moffett

Science Lecture Hall, Science Building

Thursday, September 12, 2013

12:30 – 1:20 pm

Past Seminars

“How sleep affects the developmental learning of bird song” by Kyle Jenkins

“Quantitative structure-activity relationship (QSAR) modeling of serotonin type-6 (5-HT6) and antagonists” by Daniel Russo

Science Lecture Hall, Science Building
Thursday, May 2, 2013
12:30 – 1:20 pm

“The fruit fly:  A tractable system to study tolerance of low temperatures” by Daniel Ricketti

“Mapping the distribution of regulatory sequences during Drosophila oogenesis” by Nicole Pope

Science Lecture Hall, Science Building
Thursday, April 25, 2013
12:30 – 1:20 pm

Dr. Heinz Schättler, Dept. of Electrical and Systems Engineering, Washington University

Mathematical Models for Cancer Treatments –  The Role of the Vasculature and the Immune System in Optimal Protocols for Cancer Therapies

(joint research with Urszula Ledzewicz, Southern Illinois University)

Thursday, March 28th, 2013, 12:30pm – 1:20pm, Science Bldg Lecture Hall

A systematic study of cancer treatments requires that we take into account not only the tumor and its growth, but also its microenvironment which comprises the cancerous cells, (sensitive and resistant to the treatment), healthy cells, tumor vasculature, immune system and more. In this talk, I will discuss some mathematical models that include increasingly more complex aspects of the tumor microenvironment such as tumor heterogeneity, angiogenic signaling, and tumor immune system interactions. These models will be analyzed from a dynamical systems point of view in the context of the optimal control problem of designing treatment protocols. Using methods of geometric optimal control, syntheses of optimal solutions will be described for some of these models. As more and more aspects of the tumor microenvironment are taken into account, optimal solutions change from bang-bang solutions (which correlate with the standard medical practice of giving chemotherapeutic agents in maximum tolerated doses) to administration schedules that favor singular controls (which administer agents at specific time varying reduced dose rates). This raises the possibility of metronomic administrations of agents (at low concentrations over prolonged periods without any major interruptions), an alternative scheduling approach that has shown some success in pediatric cancers. The talk will also address some of the mathematical challenges that arise in the analysis of these generally highly nonlinear, multi-input control systems.

Dr. Erica Johnson, Associate Professor, Biochemistry & Molecular Biology, Thomas Jefferson University

A Direct Effect of the Cellular Energy Charge on Global Levels of Modification by the Ubiquitin-like Modifier SUMO

Thursday, March 28th, 2013, 12:30pm, 401 Penn Lecture Hall

Dr. Gregory DavisAssistant Professor, Biology, Bryn Mawr College

Patterning challenges for optional sex:  the case of reproductive polyphenism in aphids

Thursday, March 7th, 2013, 12:30pm – 1:20pm, Science Building Lecture Hall

The pea aphid, Acyrthosiphon pisum, exhibits several environmentally cued, discrete, alternate phenotypes (polyphenisms) during its life cycle. In the case of the reproductive polyphenism, differences in day length determine whether mothers will produce daughters that reproduce either sexually by laying fertilized eggs (oviparous sexual reproduction), or asexually by allowing oocytes to complete embryogenesis within the mother without fertilization (viviparous parthenogenesis). Oocytes and embryos that are produced asexually develop more rapidly, are yolk-free, and much smaller than oocytes and embryos that are produced sexually. Perhaps most striking, the process of oocyte differentiation is truncated in the case of asexual/viviparous development, potentially precluding interactions between the oocyte and surrounding follicle cells that might take place during sexual/oviparous development. Given the important patterning roles that oocyte-follicle cell interactions play in Drosophila, these overt differences suggest that there may be underlying differences in the molecular mechanisms of pattern formation. We have found differences in the expression of torso-like, as well as activated MAP kinase, suggesting that there are important differences in the hemipteran version of the terminal patterning system between viviparous and oviparous development. Establishing such differences in the expression of patterning genes between these developmental modes is a first step toward understanding how a single genome manages to direct patterning events in such different embryological contexts.